AI overview (actually Iasper)

Histology and cellular anatomy of skeletal muscle cells

intercalated disk has gap junctions and desmosomes this allows attachment and passage of electrical pulses

actin myosin

calcium binds to troponin and tropomyosin uncouples from actin active site

excitation contraction coupling

excitation contraction

electrical impulses lead to extracellular ca2+ intake (25% of needed ca) and release of ca 2+ from sacrolemma (75% of needed Ca)

calcium is released form the sarcolemma by ca 2+ ryr uses calcium as a trigger to release calcium

high density around L-type ca channels in the plasma membrane

relaxation restitution

we also pump calcium out of the cell with a series of pumps and exchangers as well as back into the sr

1 SERCA pumps calcium into SR PLN tends to inhibit SERCA-2 while it is dephosphorylated

2 Sodium calcium exchange pump

3 calcium ATPase

Changing the pump

  1. make heart a better pump (improve pumping action)
    1. SNS/ contractility/ positive inotropic effect
  2. improve operating conditions
    1. starlings law, filling / output

Positive inotropic changes

aderingeric sympathetics sympathetic contraction

  1. binding of NA to b1 adrenoceptors in cardiac muscle cell
  2. increased concentration of cAMP
  3. activation of protein kinase A
  4. PKA phosphorylates Ltype calcium channels and RyR channel (and phospholamban?)
  5. more ca2+ ion enter per impulse