Relatively straightforward lecture just talks about TB which is great

Objectives

  • Discuss the epidemiology of mycobacterium tuberculosis
  • Understand the disease caused yb M tuberculosis
  • Discuss the host pathogen interaction in tuber culosis
  • Discuss how to diagnose active and latent m tuberculosis infection

Context

Tb is a historical killer Mortality has been reducing since 1900s TB is still really problematic in africa and asia

Tuberculosis


Mycobacterium tuberculosis

Acid fast bacillis Rich is call wall lipids, perticularly mycolic acid (which is hydrophobic). Slow growing. faculative intracellular pathoogen

Tuberculosis complex

7 bacteria which transmit tuberculosis

one is mycobacteria bovis

Transmission of TB

mainly spread through aerosols. It is good at this

Pathogenesis

Environmental exposure leads to infection which can lead to containment and persistance (latent infection) which can lead to reactivation

from infection 5-10% lead to primary tuberculosis from reactiation is also known as miliary TB (extra pulmonary TB)

Infection

Macrophage try to phagocytose the tuberculosis bacterium and then live and reproduce inside the cell

Granulomas

Granulomas form to contain the tuberculosis these force the Tb to be in stressed state which switches them to low power mode

however granlomas can become necrotic and rupture

An aside on CD4+ cells

Th1 cells produce IFNy and TNF alpha which activate macrophages TH17 calls produce IL-17 which activate neutrophils

CD8+ cells can kill infected macrophages

The IFNy can cause the macrophages to successfully consume tuberculosis

Laboratory diagnosis of tuberculosis

acid fast stain how you stain for TB

new method is a machine that just detects it in sputum samples the machina also tests for ampcillin resistance

Gold standard is culture in solid or automated liquic cultures

Susceptibility testing on cultures are really useful for figuring out what to use

Treatment of TB

multiple drugs to prevent resistance

treatment for at least 6-9 months dormant M tuberculosis necessitates prolonged treatment,

This is a highly protocolised disease

Barriers to therapy

long treatment duration complex regiment toxic effects in combination with antiretrovirals drug resistant TB and XDR TB

Treatment fo MDR/RR/preXDr and XDR tb is bedaquiline + pretomanid + linezolid+ moxifloxacin

Testing for latent TB infection

interferon famma release assay

tuberculin skin test inject TB protein and watch for reaction

Why test for thi? Identify individuals at increased risk for developing active TB